Download e-book for kindle: AIDS, Drugs of Abuse, and the Neuroimmune Axis by Herman Friedman (auth.), Herman Friedman, Toby K.
By Herman Friedman (auth.), Herman Friedman, Toby K. Eisenstein, John Madden, Burt M. Sharp (eds.)
This quantity represents the court cases of the Symposium on AIDS, medications of Abuse and the Neuroimmune Axis. This assembly was once held in San Diego, California, November 11-13, 1995. As within the prior symposia during this sequence, efficient stories have been reviewed in regards to the dating among the anxious and the immune structures about the dating among medications of abuse and infections, particularly infections through the immunode ficiency virus that motives AIDS. in recent times, a variety of investigators have started to explain the function of illicit medicines and their endogenous opposite numbers at the brain-immune axis. it's widely known that the neuroendocrine process is in detail fascinated about the consequences and manifestations of the interactions of gear of abuse and the immune procedure. The assembly on which the chapters during this ebook are dependent introduced jointly many organic scientists from an array of varied medical disciplines whose paintings is targeted at the results of gear of abuse at the neuroendocrine-immune axis and its relationships to immunodeficiency as a result of the AIDS virus. As some time past, the symposium used to be distinctive in concentrating on the . brain-immune axis from the point of view of gear of abuse instead of from the point of view of immunity or the mind itself.
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Additional info for AIDS, Drugs of Abuse, and the Neuroimmune Axis
91:161-169. 17. T. A. 1985 Cellular mechanisms of opioid tolerance: studies in single brain neurons. Mol. , 2:633-638. 18. L. E. Chronic treatment with morphine and ethanol, but not cocaine, attenuates IL-I ~ activation of Fos expression in the rat hypothalamic paraventricular nucleus. In: The Brain Immune Axis and Substance Abuse, B. Sharp, et. , Plenum Press, New York, 1995, pp. 201-208. o 42 S. L. Chang et al. 19. A. M. 1985 Interleukin-I reduces opioid binding in guinea pig brain. Peptides, 6:1149-1153.
C. (1994) Nitric oxide production and neurotoxicity mediated by activated microglia from human versus mouse brain. J. Infect. Dis. 170: 457-460. 10. Chao, C. , and Hu, S. (1994) Tumor necrosis factor-a potentiates glutamate neurotoxicity in human fetal brain cell cultures. Dev. Neurosci. 16: 172-179. II. Chao, C. , Sheng, W. , and Peterson, P. K. (1994) Priming effect of morphine on the production of tumor necrosis factor-a by microglia: implications in respiratory burst activity and human immunodeficiency virus-l expression.
In previous studies, the 6-glucuronide but not the 3-glucuronide metabolite of morphine was shown to bind to the 113 receptor; also, the potent opiate fentanyl did not bind (3, 5). Similar results were obtained for the astrocyte, brain and retinal preparations examined in the present study. The inactivity of drugs such as fentanyl and the opioid peptides had been utilized previously as well as in the present study to distinguish the 113 receptor from other receptor SUbtypes. This was of particular importance for studies of brain where classical opioid receptor subtypes are routinely also present.
AIDS, Drugs of Abuse, and the Neuroimmune Axis by Herman Friedman (auth.), Herman Friedman, Toby K. Eisenstein, John Madden, Burt M. Sharp (eds.)